bims-smemid Biomed News
on Stress metabolism in mitochondrial dysfunction
Issue of 2024‒04‒21
two papers selected by
Deepti Mudartha, The International Institute of Molecular Mechanisms and Machines



  1. J Cell Biol. 2024 May 06. pii: e202403190. [Epub ahead of print]223(5):
      Using an engineered mitochondrial clogger, Krakowczyk et al. (https://doi.org/10.1083/jcb.202306051) identified the OMA1 protease as a critical component that eliminates import failure at the TOM translocase in mammalian cells, providing a novel quality control mechanism that is distinct from those described in yeast.
    DOI:  https://doi.org/10.1083/jcb.202403190
  2. Bioessays. 2024 Apr 14. e2300218
      Dietary methionine restriction (MR) is associated with a spectrum of health-promoting benefits. Being conducive to prevention of chronic diseases and extension of life span, MR can activate integrated responses at metabolic, transcriptional, and physiological levels. However, how the mitochondria of MR influence metabolic phenotypes remains elusive. Here, we provide a summary of cellular functions of methionine metabolism and an overview of the current understanding of effector mechanisms of MR, with a focus on the aspect of mitochondria-mediated responses. We propose that mitochondria can sense and respond to MR through a modulatory role of lipoylation, a mitochondrial protein modification sensitized by MR.
    Keywords:  cellular metabolism; lipidomics; metabolomics; methioinine metabolism; phospholipid
    DOI:  https://doi.org/10.1002/bies.202300218